Laserfiche WebLink
4.1 – Air Quality <br />Draft Environmental Impact Report February 2021 <br />14800 W. Schulte Road Logistics Center 4.1-6 <br />Long-term (months to years) exposure to PM2.5 has been linked to premature death, particularly in people who have <br />chronic heart or lung diseases, and reduced lung function growth in children. The effects of long-term exposure to <br />PM10 are less clear, although several studies suggest a link between long-term PM10 exposure and respiratory <br />mortality. The International Agency for Research on Cancer published a review in 2015 that concluded that PM in <br />outdoor air pollution causes lung cancer (CARB 2017). <br />People with influenza, people with chronic respiratory and cardiovascular diseases, and older adults may suffer <br />worsening illness and premature death as a result of breathing PM. People with bronchitis can expect aggravated <br />symptoms from breathing PM. Children may experience a decline in lung function due to breathing in PM 10 and <br />PM2.5 (EPA 2009). <br />PM encompasses a physically and chemically diverse class of ambient air pollutants of both anthropogenic and <br />biological origin. The PM standard is the only National Ambient Air Quality Standard (NAAQS) that does not target a <br />specific chemical or family of chemical species (NRC 2005). The range of human health effects associated with <br />ambient PM levels or demonstrated in laboratory studies has expanded from earlier concerns for total mortality and <br />respiratory morbidity to include cardiac mortality and morbidity, blood vessel constriction, stroke, premature birth, <br />low birth weight, retarded lung growth, enhancement of allergic responses, reduced resistance to infection, <br />degenerative lesions in the brain, and lung cancer (EPA 2004). <br />Lead. Lead in the atmosphere occurs as particulate matter. Sources of lead include leaded gasoline; the <br />manufacturing of batteries, paints, ink, ceramics, and ammunition; and secondary lead smelters. Prior to 1978, <br />mobile emissions were the primary source of atmospheric lead. Between 1978 and 1987, the phaseout of leaded <br />gasoline reduced the overall inventory of airborne lead by nearly 95%. With the phaseout of leaded gasoline, <br />secondary lead smelters, battery recycling, and manufacturing facilities are becoming lead -emissions sources of <br />greater concern. <br />Prolonged exposure to atmospheric lead poses a serious threat to human health. Health effects associated with <br />exposure to lead include gastrointestinal disturbances, anemia, kidney disease, and, in severe cases, <br />neuromuscular and neurological dysfunction. Of particular concern are low-level lead exposures during infancy and <br />childhood. Such exposures are associated with decrements in neurobehavioral performance, including intelligence <br />quotient performance, psychomotor performance, reaction time, and growth. Children are highly susceptible to the <br />effects of lead. <br />Reactive Organic Gases. Hydrocarbons are organic gases that are formed from hydrogen and carbon and <br />sometimes other elements. Hydrocarbons that contribute to formation of O3 are referred to and regulated as ROGs <br />(also referred to as VOCs). Combustion engine exhaust, oil refineries, and fossil-fueled power plants are the sources <br />of hydrocarbons. Other sources of hydrocarbons include evaporation from petroleum fuels, solvents, dry cleaning <br />solutions, and paint. <br />The primary health effects of ROGs result from the formation of O 3 and its related health effects. High levels of <br />ROGs in the atmosphere can interfere with oxygen intake by reducing the amount of available oxygen through <br />displacement. Carcinogenic forms of hydrocarbons, such as benzene, are considered TACs. There are no separate <br />health standards for ROGs as a group.