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SU0014502
Environmental Health - Public
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SU0014502
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Entry Properties
Last modified
8/25/2022 10:29:54 AM
Creation date
11/4/2021 4:18:13 PM
Metadata
Fields
Template:
EHD - Public
ProgramCode
2600 - Land Use Program
RECORD_ID
SU0014502
PE
2675
FACILITY_NAME
PA-1900240
STREET_NUMBER
11000
Direction
N
STREET_NAME
WEST
STREET_TYPE
LN
City
LODI
Zip
95242-
APN
05908029, -07 -30
ENTERED_DATE
11/4/2021 12:00:00 AM
SITE_LOCATION
11000 N WEST LN
RECEIVED_DATE
5/2/2022 12:00:00 AM
P_LOCATION
99
P_DISTRICT
004
QC Status
Approved
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EHD - Public
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Administrative Draft Environmental Impact Report <br /> Gill Medical Center Project <br /> Carbon Monoxide <br /> CO in the urban environment is associated primarily with the incomplete combustion of fossil fuels in <br /> motor vehicles. CO combines with hemoglobin in the bloodstream and reduces the amount of oxygen <br /> that can be circulated through the body. High CO concentrations can cause headaches, aggravate <br /> cardiovascular disease and impair central nervous system functions. CO concentrations can vary greatly <br /> over comparatively short distances. Relatively high concentrations of CO are typically found near crowded <br /> intersections and along heavy roadways with slow moving traffic. Even under the most severe <br /> meteorological and traffic conditions, high concentrations of CO are limited to locations within relatively <br /> short distances of the source. Overall CO emissions are decreasing as a result of the Federal Motor Vehicle <br /> Control Program, which has mandated increasingly lower emission levels for vehicles manufactured since <br /> 1973. <br /> Nitrogen Oxides <br /> Nitrogen gas comprises about 80 percent of the air and is naturally occurring.At high temperatures and <br /> under certain conditions, nitrogen can combine with oxygen to form several different gaseous <br /> compounds collectively called nitric oxides (NOx). Motor vehicle emissions are the main source of NOx in <br /> urban areas. NOx is very toxic to animals and humans because of its ability to form nitric acid with water in <br /> the eyes, lungs, mucus membrane, and skin. In animals, long-term exposure to NOx increases <br /> susceptibility to respiratory infections, and lowering resistance to such diseases as pneumonia and <br /> influenza. Laboratory studies show that susceptible humans, such as asthmatics,who are exposed to high <br /> concentrations can suffer from lung irritation or possible lung damage. Precursors of NOx, such as NO and <br /> NO2, attribute to the formation of 03 and PM2.5. Epidemiological studies have also shown associations <br /> between NO2 concentrations and daily mortality from respiratory and cardiovascular causes and with <br /> hospital admissions for respiratory conditions. <br /> Ozone <br /> 03 is a secondary pollutant, meaning it is not directly emitted. It is formed when volatile organic <br /> compounds (VOCs) or ROGs and NOx undergo photochemical reactions that occur only in the presence of <br /> sunlight.The primary source of ROG emissions is unburned hydrocarbons in motor vehicle and other <br /> internal combustion engine exhaust. NOx forms as a result of the combustion process, most notably due <br /> to the operation of motor vehicles. Sunlight and hot weather cause ground-level 03 to form. Ground-level <br /> 03 is the primary constituent of smog. Because 03 formation occurs over extended periods of time, both <br /> 03 and its precursors are transported by wind and high 03 concentrations can occur in areas well away <br /> from sources of its constituent pollutants. <br /> People with lung disease, children, older adults, and people who are active can be affected when 03 levels <br /> exceed ambient air quality standards. Numerous scientific studies have linked ground-level 03 exposure to <br /> a variety of problems including lung irritation, difficult breathing, permanent lung damage to those with <br /> repeated exposure, and respiratory illnesses. <br /> Air Quality 4.5-3 October 2021 <br />
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