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SU0004818 SSCRPT
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SU0004818 SSCRPT
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Last modified
5/7/2020 11:31:14 AM
Creation date
9/5/2019 10:45:54 AM
Metadata
Fields
Template:
EHD - Public
ProgramCode
2600 - Land Use Program
FileName_PostFix
SSCRPT
RECORD_ID
SU0004818
PE
2622
FACILITY_NAME
PA-0500035
STREET_NUMBER
19300
Direction
W
STREET_NAME
GRANT LINE
STREET_TYPE
RD
City
TRACY
APN
20906011, &
ENTERED_DATE
2/9/2005 12:00:00 AM
SITE_LOCATION
19300 W GRANT LINE RD
RECEIVED_DATE
2/1/2005 12:00:00 AM
P_LOCATION
99
P_DISTRICT
005
QC Status
Approved
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SJGOV\rtan
Supplemental fields
FilePath
\MIGRATIONS\G\GRANT LINE\19300\PA-0500035\SU0004818\SSC RPT.PDF
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EHD - Public
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L ' <br /> ti TUSO PROPERTY Page 5 <br /> �- WKA No. 5475.02 <br /> June 2, 2003 <br /> 1~ <br /> i Regarding EMFs on the subject property,we have also included below the Conclusions section out <br /> ` of the Executive Summary from the May 1999 National Institutes of Health(NIH),National <br /> Institute of Environmental Health Sciences(NIEHS) "Report on Health Effects from Exposure to <br /> LPowerline Frequency EMFs, "NIH Publication No. 99-4493. We did not review the entire report <br /> nor was the body of the report readily available for us to review. The NIH report is excerpted as <br /> follows: <br /> ` "The scientific evidence suggesting that EMF exposures pose any health risk is weak. <br /> The strongest evidence for health effects comes from associations observed in human <br /> populations with two forms of cancer: childhood leukemia and chronic lymphocytic <br /> leukemia in occupationally exposed adults. While the support from individual studies is <br /> L weak, the epidemiological studies demonstrate for some methods of measuring exposure, <br /> a fairly consistent pattern of a small, increased risk with increasing exposure that is <br /> L somewhat weaker for chronic lymphocytic leukemia than for childhood leukemia. In <br /> contrast, the mechanistic studies and the animal toxicology literature fail to demonstrate <br /> any consistent pattern across studies although sporadic findings of biological effects <br /> (including increased cancers in animals)have been reported. No indication of increased <br /> L <br /> leukemias in experimental animals has been observed. <br /> "The lack of connection between the human data and experimental data(animal and <br /> mechanistic) severely complicates the interpretation of these results. The human data are <br /> L in the `right' species, are tied to `real-life' exposures and show some consistency that is <br /> difficult to ignore. This assessment is tempered by the observation that given the weak <br /> ` magnitude of these increased risks, some other factor or common source of error could <br /> explain these findings. However, no consistent explanation other than exposure to EMFs <br /> has been identified. <br /> i "Epidemiological studies have serious limitations in their ability to demonstrate a cause <br /> �- and effect relationship whereas laboratory studies,by design, can clearly show that cause <br /> and effect are possible. Virtually all of the laboratory evidence in animals and humans <br /> and most of the mechanistic work done in cells fail to support a causal relationship <br /> between exposure to EMFs at environmental levels and changes in biological function or <br /> V <br /> WALLACE•KUHL <br /> &ASSOCIATES INC. <br />
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